Ingredient intelligence

Niacinamide

Skin barrier · Niacinamide · C6H6N2O

Niacinamide is a form of vitamin B3 involved in cellular metabolism as a precursor of NAD+. It has been studied for barrier-related, pigmentation-related and sebum-related endpoints in skin.

Molecular identity

Two-dimensional chemical structure of Niacinamide
Formula
C6H6N2O
Molecular weight
122.12 g/mol
PubChem CID
936
ChEBI
CHEBI:17154
Open PubChem record →

Biological targets

  • Nicotinamide phosphoribosyltransferase (NAMPT)

    high confidence

    targets · enzyme

    Rate-limiting enzyme of the NAD salvage pathway that uses nicotinamide as substrate.

    UniProt P43490
  • Poly(ADP-ribose) polymerase 1 (PARP1)

    medium confidence

    inhibits · enzyme

    Nicotinamide is a product-inhibitor of PARP enzymes in biochemical assays.

    UniProt P09874
  • Sirtuin 1 (SIRT1)

    medium confidence

    modulates · enzyme

    UniProt Q96EB6
  • ADP-ribosyl cyclase CD38

    low confidence

    inhibits · enzyme

    UniProt P28907

Molecules & genes

Molecules

  • NicotinamideC6H6N2O · 122.12 g/mol
  • NAD+ (nicotinamide adenine dinucleotide)C21H27N7O14P2 · 663.43 g/mol

Genes / proteins

How could this ingredient interact with biology?

Biological network

Ingredient → molecule → target → gene/protein → pathway → process → studied outcome. Relationship strength is drawn from the curated confidence tier; nothing is added to make the chain look complete.

100%
NiacinamideNicotinamideNAD+ (nicotinamide adenin…Nicotinamide phosphoribos…Poly(ADP-ribose) polymera…Sirtuin 1 (SIRT1)NAMPTPARP1SIRT1Nicotinate metabolism (NA…NAD+ dependent deacetylat…Keratinocyte differentiat…Cellular energy metabolismEpidermal barrier formati…Melanosome transferStudied for barrier suppo…Studied for reduction of …Studied for visible signs…

drag to pan · hover to isolate a path · click a node for detail

IngredientMoleculeTargetGenePathwayProcessOutcome

3 lower-confidence relationships are hidden at this filter level. Use “Show more biology” to include them.

Text alternative for this network
  • Niacinamide is molecule Nicotinamidehigh confidence
  • Niacinamide is molecule NAD+ (nicotinamide adenine dinucleotide)high confidence
  • Nicotinamide targets Nicotinamide phosphoribosyltransferase (NAMPT)high confidence
  • Nicotinamide inhibits Poly(ADP-ribose) polymerase 1 (PARP1)medium confidence
  • Nicotinamide modulates Sirtuin 1 (SIRT1)medium confidence
  • Nicotinamide phosphoribosyltransferase (NAMPT) encoded by NAMPThigh confidence
  • Poly(ADP-ribose) polymerase 1 (PARP1) encoded by PARP1medium confidence
  • Sirtuin 1 (SIRT1) encoded by SIRT1medium confidence
  • NAMPT participates in Nicotinate metabolism (NAD salvage)high confidence
  • PARP1 participates in Nicotinate metabolism (NAD salvage)medium confidence
  • SIRT1 participates in Nicotinate metabolism (NAD salvage)medium confidence
  • NAMPT participates in NAD+ dependent deacetylation (sirtuins)medium confidence
  • PARP1 participates in NAD+ dependent deacetylation (sirtuins)medium confidence
  • SIRT1 participates in NAD+ dependent deacetylation (sirtuins)medium confidence
  • NAMPT participates in Keratinocyte differentiationmedium confidence
  • PARP1 participates in Keratinocyte differentiationmedium confidence
  • SIRT1 participates in Keratinocyte differentiationmedium confidence
  • Nicotinate metabolism (NAD salvage) associated with Cellular energy metabolismhigh confidence
  • NAD+ dependent deacetylation (sirtuins) associated with Cellular energy metabolismmedium confidence
  • Keratinocyte differentiation associated with Cellular energy metabolismmedium confidence
  • Nicotinate metabolism (NAD salvage) associated with Epidermal barrier formationmedium confidence
  • NAD+ dependent deacetylation (sirtuins) associated with Epidermal barrier formationmedium confidence
  • Keratinocyte differentiation associated with Epidermal barrier formationmedium confidence
  • Nicotinate metabolism (NAD salvage) associated with Melanosome transfermedium confidence
  • NAD+ dependent deacetylation (sirtuins) associated with Melanosome transfermedium confidence
  • Keratinocyte differentiation associated with Melanosome transfermedium confidence
  • Cellular energy metabolism studied in Studied for barrier support and transepidermal water lossmedium confidence
  • Epidermal barrier formation studied in Studied for barrier support and transepidermal water lossmedium confidence
  • Melanosome transfer studied in Studied for barrier support and transepidermal water lossmedium confidence
  • Cellular energy metabolism studied in Studied for reduction of uneven pigmentationmedium confidence
  • Epidermal barrier formation studied in Studied for reduction of uneven pigmentationmedium confidence
  • Melanosome transfer studied in Studied for reduction of uneven pigmentationmedium confidence
  • Cellular energy metabolism studied in Studied for visible signs of skin ageingmedium confidence
  • Epidermal barrier formation studied in Studied for visible signs of skin ageingmedium confidence
  • Melanosome transfer studied in Studied for visible signs of skin ageingmedium confidence

Biological pathways

Nicotinate metabolism (NAD salvage)

high confidence

Nicotinamide is recycled into NAD+ by NAMPT and NMNAT enzymes, supplying redox and signalling cofactors.

Reactome R-HSA-196807

NAD+ dependent deacetylation (sirtuins)

medium confidence

NAD+ availability constrains sirtuin-mediated protein deacetylation.

Reactome R-HSA-3247509

Keratinocyte differentiation

medium confidence

Barrier-protein expression programme examined in niacinamide-treated keratinocyte models.

Gene Ontology GO:0030216

Evidence engine

Moderate

Some human evidence exists, but replication or sample sizes are limited.

Skinceptor Evidence Index

60

experimental · of 100

This index is a research-navigation aid describing how much evidence exists and how it was produced. It is not a medical safety score, an efficacy guarantee, or a clinical recommendation.

Study composition

  • Human2
  • In-vitro2
  • Review1
Total studies
5
Human studies
2
Largest sample
50
Range
2000–2018

Index breakdown

  • Study design quality47% × 28%

    Mean design weight across curated studies (meta-analysis 1.0, RCT 0.9, other human 0.7, review 0.45, animal 0.35, in-vitro 0.25).

  • Human evidence share67% × 22%

    Proportion of curated evidence measured in people rather than cells or animals.

  • Evidence volume82% × 14%

    Log-scaled count of curated studies, so volume alone cannot dominate the score.

  • Largest sample size57% × 12%

    Log-scaled largest reported sample size across curated human studies.

  • Replication67% × 12%

    Independent human studies, capped at three; one study is never treated as replicated.

  • Recency60% × 7%

    Linear decay over a twenty-year window from the most recent curated publication.

  • Source traceability40% × 5%

    Share of curated studies with a verified PMID or DOI in this dataset.

How is this determined? →

Insufficient comparable evidence for quantitative synthesis.

Research timeline

How the evidence accumulated

  1. 2000

    Topical niacinamide and epidermal barrier lipid synthesis in cultured keratinocytes

    In-vitrosample size unavailable · Journal of Cosmetic Science

  2. 2002

    The effect of niacinamide on reducing cutaneous pigmentation and suppression of melanosome transfer

    Humann = 18 · British Journal of Dermatology

  3. 2005

    Niacinamide: a B vitamin that improves aging facial skin appearance

    Humann = 50 · Dermatologic Surgery

  4. 2011

    Nicotinamide as an inhibitor of poly(ADP-ribose) polymerase activity

    In-vitrosample size unavailable · Biochemical Journal

  5. 2018

    Nicotinamide and NAD precursor metabolism in human cells

    Reviewsample size unavailable · Cell Metabolism (review)

Ingredient fingerprint

21 nodes · 46 relationships · seed "niacinamide"

The fingerprint is a deterministic visual signature: ridge amplitude comes from the number of entities in each tier, node placement from identifier hashes, and opacity from confidence. Two ingredients can never produce the same figure unless their networks are identical.

Open in Biology as Art →

Research

5 curated studies

ReviewCell Metabolism (review) · 2018

Nicotinamide and NAD precursor metabolism in human cells

Summarises the NAD salvage route in which nicotinamide is converted by NAMPT and NMNAT enzymes to NAD+, supporting redox and signalling functions.

LimitationsReview of mechanism, largely non-cutaneous; does not evaluate topical delivery.

In-vitroBiochemical Journal · 2011

Nicotinamide as an inhibitor of poly(ADP-ribose) polymerase activity

Characterises nicotinamide as a weak product inhibitor of PARP enzymes in cell-free assays.

LimitationsBiochemical assay; inhibitory concentrations far above physiological skin levels.

HumanDermatologic Surgery · 2005n = 50

Niacinamide: a B vitamin that improves aging facial skin appearance

Reported improvements in several appearance endpoints including fine lines and skin texture over 12 weeks of use versus vehicle.

LimitationsManufacturer-affiliated authorship; appearance endpoints partly investigator-graded.

PMID 16029679Niacinamide
HumanBritish Journal of Dermatology · 2002n = 18

The effect of niacinamide on reducing cutaneous pigmentation and suppression of melanosome transfer

Reported reduced hyperpigmentation with topical niacinamide, and reduced melanosome transfer to keratinocytes in an accompanying co-culture model.

LimitationsSmall sample, short duration, split-face design without long-term follow-up.

PMID 12100180Niacinamide
In-vitroJournal of Cosmetic Science · 2000

Topical niacinamide and epidermal barrier lipid synthesis in cultured keratinocytes

Reported increased ceramide and free fatty acid synthesis in keratinocyte culture after niacinamide exposure.

LimitationsIn-vitro only; concentrations may not reflect in-use exposure.

Community evidence

Submitted by readers

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Reader submissions appear here only after review. They are not part of the curated dataset and do not affect the Skinceptor Evidence Index.

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Sources